Search results for "Blocking antibody"

showing 10 items of 11 documents

7-O-acetyl-GD3 in human T-lymphocytes is detected by a specific T-cell-activating monoclonal antibody.

1995

The monoclonal antibody U5, which is a potent inducer of proliferation in human T-cells, was found to bind to an alkali-sensitive derivative of ganglioside GD3. Using immunochemical and spectroscopic methods, the structure of the U5 antigen was determined as 7-O-acetyl-GD3. The antibody U5 did not react with 9-O-acetyl-GD3 and bound severalfold more stronger to 7-O-acetyl-GD3 than to GD3. U5 is the first antibody known to detect preferentially 7-O-acetyl-GD3. Flow cytometric analysis showed that each major class of human leukocytes contained a significant fraction of cells binding the U5 antibody.

medicine.drug_classT cellT-LymphocytesImmunoblottingMolecular Sequence DataMonoclonal antibodyLymphocyte ActivationBiochemistryMass SpectrometryAntigenAntibody SpecificityGangliosidesBlocking antibodymedicineLeukocytesGanglioside GD3AnimalsHumansMolecular BiologyDirect fluorescent antibodybiologyChemistryAntibodies MonoclonalCell BiologyMolecular biology7-O-acetyl-GD3Kineticsmedicine.anatomical_structureMilkCarbohydrate Sequencebiology.proteinlipids (amino acids peptides and proteins)CattleFemaleAntibodyThe Journal of biological chemistry
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Novel Lymphocyte-Independent Antitumor Activity by PD-1 Blocking Antibody against PD-1+ Chemoresistant Lung Cancer Cells

2022

Abstract Purpose: Antibodies against the lymphocyte PD-1 (aPD-1) receptor are cornerstone agents for advanced non–small cell lung cancer (NSCLC), based on their ability to restore the exhausted antitumor immune response. Our study reports a novel, lymphocyte-independent, therapeutic activity of aPD-1 against NSCLC, blocking the tumor-intrinsic PD-1 receptors on chemoresistant cells. Experimental Design: PD-1 in NSCLC cells was explored in vitro at baseline, including stem-like pneumospheres, and following treatment with cisplatin both at transcriptional and protein levels. PD-1 signaling and RNA sequencing were assessed. The lymphocyte-independent antitumor activity of aPD-1 was explored in…

chemoresistantCancer ResearchOncologylung cancer cellsAnti–PD-1 Lymphocyte-Independent Activity against NSCLClymphocyte-independent PD-1 blocking antibody against PD-1+ chemoresistant lung cancer cellslymphocyte-independentPD-1 blocking antibody against PD-1+Clinical Cancer Research
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Thyrotropin Receptor Blocking Antibodies.

2018

AbstractAutoantibodies (Ab) against the thyroid-stimulating hormone receptor (TSHR) are frequently found in autoimmune thyroid disease (AITD). Autoantibodies to the TSHR (anti-TSHR-Ab) may mimic or block the action of TSH or be functionally neutral. Measurement of anti-TSHR-Ab can be done either via competitive-binding immunoassays or with functional cell-based bioassays. Antibody-binding assays do not assess anti-TSHR-Ab functionality, but rather measure the concentration of total anti-TSHR binding activity. In contrast, functional cell-based bioassays indicate whether anti-TSHR-Ab have stimulatory or blocking activity. Historically bioassays for anti-TSHR-Ab were research tools and were u…

medicine.medical_specialtyendocrine systemendocrine system diseasesEndocrinology Diabetes and MetabolismGraves' diseaseClinical Biochemistry030209 endocrinology & metabolismHashimoto DiseaseReviewBiochemistryThyroiditisThyrotropin receptor03 medical and health sciences0302 clinical medicineEndocrinologyInternal medicineBlocking antibodymedicineAnimalsHumansReceptorAntibodies BlockingAutoantibodiesbinding assaycell-based bioassaybiologybusiness.industryBiochemistry (medical)AutoantibodyReceptors ThyrotropinGeneral MedicineHashimoto’s thyroiditismedicine.diseaseTSH receptor blocking autoantibodieseye diseasesEndocrinologyHormone receptor030220 oncology & carcinogenesisImmunologybiology.proteinBiological AssayAntibodybusinessGraves’ diseasehormones hormone substitutes and hormone antagonistsHormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme
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Novel Microglia Depletion Systems: A Genetic Approach Utilizing Conditional Diphtheria Toxin Receptor Expression and a Pharmacological Model Based on…

2019

Microglia are the main population of macrophage residing in the central nervous system (CNS). Depletion experiments gave important insights into the physiology and function of microglia in healthy and diseased CNS. Ablation of microglia can be achieved by application of pharmacological or genetic tools. Here, we describe two approaches to ablate microglia: an efficient genetic model that utilizes DTRMG mouse line that has diphtheria toxin receptor (DTR) expression regulated by the promoter activity of the fractalkine receptor (CX3CR1) gene, and a pharmacological model that utilizes the blocking of macrophage colony-stimulating factor 1 receptor (CSF-1R) with a blocking antibody. Both the ad…

0301 basic medicineDiphtheria toxinMacrophage Colony-Stimulating Factor 1 Receptoreducation.field_of_studyMicrogliaPopulationBiologyCell biology03 medical and health sciences030104 developmental biology0302 clinical medicinemedicine.anatomical_structureGenetic modelBlocking antibodyCX3CR1medicineeducationReceptor030217 neurology & neurosurgery
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Prevalence and clinical relevance of thyroid stimulating hormone receptor-blocking antibodies in autoimmune thyroid disease

2017

Summary The prevalence and clinical relevance of thyroid stimulating hormone (TSH) receptor (TSHR) blocking antibodies (TBAb) in patients with autoimmune thyroid disease (AITD) was investigated. Serum TBAb were measured with a reporter gene bioassay using Chinese hamster ovary cells. Blocking activity was defined as percentage inhibition of luciferase expression relative to induction with bovine TSH alone (cut-off 40% inhibition). All samples were measured for TSHR stimulatory antibody (TSAb) and TSHR binding inhibiting immunoglobulins (TBII). A total of 1079 unselected, consecutive patients with AITD and 302 healthy controls were included. All unselected controls were negative for TBAb and…

AdultMaleendocrine systemmedicine.medical_specialtyAdolescentendocrine system diseasesGraves' diseaseImmunologyThyroid Gland030209 endocrinology & metabolismCHO CellsHashimoto DiseaseThyroiditisYoung Adult03 medical and health sciencesCricetulus0302 clinical medicineCricetinaeInternal medicineBlocking antibodyPrevalencemedicineAnimalsHumansImmunology and AllergyEuthyroidClinical significanceAutoantibodiesbiologybusiness.industryChinese hamster ovary cellThyroidThyroiditis AutoimmuneReceptors ThyrotropinOriginal ArticlesMiddle Agedmedicine.diseaseeye diseasesGraves Diseasemedicine.anatomical_structureEndocrinology030220 oncology & carcinogenesisImmunologybiology.proteinBiological AssayFemaleAntibodybusinesshormones hormone substitutes and hormone antagonistsClinical and Experimental Immunology
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Analytical Performance and Validation of a Bioassay for Thyroid-Blocking Antibodies

2016

A cell-based bioassay for the measurement of thyroid blocking autoantibodies (TBAb) has been recently reported. The analytical performance and validation of this bioassay is assessed and described.Chinese hamster ovary cells expressing a chimeric thyrotropin receptor were treated with bovine (b) TSH and different concentrations of an immunoglobulin G (IgG) monoclonal human TBAb (K1-70). TBAb was measured as a function of luciferase activity relative to bTSH alone and expressed as percent inhibition. Results obtained in the chimeric cell line were compared with those of a wild-type cell line. Analytical performance studies were subsequently performed with the chimeric cell line only.Immunode…

Endocrinology Diabetes and MetabolismThyroid Gland030209 endocrinology & metabolismCHO CellsImmunoglobulin GThyrotropin receptor03 medical and health sciencesCricetulus0302 clinical medicineEndocrinologyBlocking antibodyAnimalsHumansBioassayAntibodies BlockingAutoantibodiesDetection limitbiologyChemistryChinese hamster ovary cellReproducibility of ResultsMolecular biologyImmunoglobulin G030220 oncology & carcinogenesisMonoclonalbiology.proteinBiological AssayAntibodyImmunoglobulins Thyroid-StimulatingThyroid
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PD-1 signalling in CD4+T cells restrains their clonal expansion to an immunogenic stimulus, but is not critically required for peptide-induced tolera…

2010

Summary The ultimate outcome of T-cell recognition of peptide–major histocompatibility complex (MHC) complexes is determined by the molecular context in which antigen presentation is provided. The paradigm is that, after exposure to peptides presented by steady-state dendritic cells (DCs), inhibitory signals dominate, leading to the deletion and/or functional inactivation of antigen-reactive T cells. This has been utilized in a variety of models providing peptide antigen in soluble form in the absence of adjuvant. A co-inhibitory molecule of considerable current interest is PD-1. Here we show that there is the opportunity for the PD-1/PD-L1 interaction to function in inhibiting the T-cell r…

CD4-Positive T-LymphocytesOvalbuminTransgeneProgrammed Cell Death 1 ReceptorImmunologyAntigen presentationMice TransgenicCell SeparationCD8-Positive T-LymphocytesBiologyLymphocyte ActivationMajor histocompatibility complexMiceImmune systemBlocking antibodyImmune ToleranceAnimalsImmunology and AllergyT-cell receptorOriginal ArticlesFlow CytometryAntigens DifferentiationPeptide FragmentsCell biologyMice Inbred C57BLTolerance inductionPhenotypeImmunologybiology.proteinCD8Signal TransductionImmunology
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Cemiplimab for locally advanced and metastatic cutaneous squamous-cell carcinomas: Real-life experience from the French CAREPI study group

2021

Although cemiplimab has been approved for locally advanced (la) and metastatic (m) cutaneous squamous-cell carcinomas (CSCCs), its real-life value has not yet been demonstrated. An early-access program enrolled patients with la/mCSCCs to receive cemiplimab. Endpoints were best overall response rate (BOR), progression-free survival (PFS), overall survival (OS), duration of response (DOR) and safety. The 245 patients (mean age 77 years, 73% male, 49% prior systemic treatment, 24% immunocompromised, 27% Eastern Cooperative Oncology Group performance status (PS) ≥ 2) had laCSCCs (35%) or mCSCCs (65%). For the 240 recipients of ≥1 infusion(s), the BOR was 50.4% (complete, 21%

Cancer Researchmedicine.medical_specialtycutaneous squamous cell carcinomaLocally advancedBest Overall Response[SDV.CAN]Life Sciences [q-bio]/CancerGastroenterologyArticle030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicineInternal medicineOverall survivalMedicineAdverse effectGroup performanceRC254-282Immune statusbusiness.industryNeoplasms. Tumors. Oncology. Including cancer and carcinogensMean agemedicine.diseasechronic dermatosisToxic epidermal necrolysis3. Good healthimmunocompromisedreal-life settingOncology030220 oncology & carcinogenesisPD-1–blocking antibodycemiplimabbusiness[SDV.MHEP.DERM]Life Sciences [q-bio]/Human health and pathology/Dermatology
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CD36-fibrin interaction propagates FXI-dependent thrombin generation of human platelets.

2019

Thrombin converts fibrinogen to fibrin and activates blood and vascular cells in thrombo-inflammatory diseases. Platelets are amplifiers of thrombin formation when activated by leukocyte- and vascular cell-derived thrombin. CD36 on platelets acts as sensitizer for molecules with damage-associated molecular patterns, thereby increasing platelet reactivity. Here, we investigated the role of CD36 in thrombin-generation on human platelets, including selected patients with advanced chronic kidney disease (CKD). Platelets deficient in CD36 or blocked by anti-CD36 antibody FA6.152 showed impaired thrombin generation triggered by thrombin in calibrated automated thrombography. Using platelets with …

0301 basic medicineBlood PlateletsCD36 AntigensCD36InflammationFibrinogenBiochemistryFibrin03 medical and health sciences0302 clinical medicineThrombinBlocking antibodyGeneticsmedicineHumansPlateletRenal Insufficiency ChronicMolecular BiologyFactor XIFibrinbiologyChemistryCell adhesion moleculeThrombinPlatelet ActivationBlood Coagulation FactorsCell biology030104 developmental biologybiology.proteinmedicine.symptom030217 neurology & neurosurgerycirculatory and respiratory physiologyBiotechnologymedicine.drugFASEB journal : official publication of the Federation of American Societies for Experimental BiologyREFERENCES
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Graves’ disease

2017

stimulating antibodiesmedicine.medical_specialtyfunctional TSH receptor antibodiesEndocrinology Diabetes and MetabolismGraves' disease030209 endocrinology & metabolismHyperthyroidismAntibodiesDiagnosis DifferentialIodine Radioisotopes03 medical and health sciencesEndocrinology0302 clinical medicineAntithyroid AgentsPregnancyInternal medicineBlocking antibodyHumansMedicineNomenclatureGeneral Commentarybusiness.industryRemission InductionReceptors Thyrotropinmedicine.diseaseGraves DiseaseAnti-thyroid autoantibodiesCausalityPregnancy Complicationsblocking antibodiesEndocrinology030220 oncology & carcinogenesisImmunologyTSH receptor antibodyThyroidectomyFemalenomenclatureGraves’ diseasebusinessFrontiers in Endocrinology
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